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NR 572/ NR572 MIDTERM EXAM: ADVANCED ACUTE
CARE MANAGEMENT GUIDE| QUESTIONS & ANSWERS|
GRADE A| 100% CORRECT (VERIFIED
SOLUTIONS)(NEW 2025/ 2026 UPDATE) -
CHAMBERLAIN
1.Adult respiratory distress syndrome (ARDS): RF due to
severe hypoxemia, resulting in lung parenchyma inflammation, a clinical syndrome of the lungs
2.ARDS cause: an inflammatory response associated with
capillary endothelial injury and diffuse alveolar damage in lungs.blood and plasma are leaked into the airspaces, hypoxemia ensues.
3.systemic inflammation due to ARDS: Inflammation in the lung
may also lead to dysfunction, or shock to the kidneys, heart, and/or muscles.
4.survivors of ARDS: varying degrees of pulmonary artery
vasoconstriction and subsequent pulmonary hypertension as long-term complications for
5.three distinctive phases of ARDS: exudative, proliferative, and
fibrotic phases are the
6.Exudative Stage: Occurs 5-7 day(systemic inflammation),
increased permeability of the alveolar-capillary barrier, formation 1 / 2
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of protein-rich, hyalin membranes along the alveolar walls, accumulation of high protein alveolar exudate within the alveolar airspaces represents noncardiogenic pulmonary edema, Extravasation of neutrophils migrate to the lung parenchyma leading to extensive alveolar damage and diffuse alveolar hemorrhage what stage
7.Proliferative Phase: 7-21 days, Fibroblast proliferation,
collagen deposition, and early fibrotic changes are observed within the pulmonary interstitial as the alveolar exudate and hyalin membranes are absorbed what phase
8.Fibrotic Phase: within several weeks, May lead to pulmonary
fibrosis ¼ of those will have significant fibrotic lung disease, restrictive ventilatory defect on PFTs, pulmonary fibrosis due to ARDS correlates with increased mortality risk
9.Etiology and Risk Factors of ARDS: common cause is
infection, intrapulmonary (ex: pneumonia) or systemic (ex: sepsis), aspiration of gastric contents, trauma, multiple blood transfusions, severe trauma, near-drowning, lung or stem cell transplant, fat embolization, metabolic acidosis, and sickle cell acute chest syndrome are
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