NUR 330 Osteoarthritis
- osteoarthritis
Answer -OA results from cartilage damage that triggers a metabolic response at level of chondrocytes
-Cartilage becomes
Dull, yellow, and granular Soft and less elastic Less able to resist wear with heavy use Single causes for OA has not been identified
- etiology and pathophysiology of OA
Answer -Inflammation not characteristics of OA
-Inflammatory change contributes to early pain and stiffness -Gradual loss of articular cartilage -Not normal part of aging process
-Cartilage destruction
Begins between ages 20 and 30, Regular exercise can help prevent, Idiopathic
-Factors that have been linked to OA
Age, Decreased estrogen at menopause, Obesity, Anterior cruciate ligament injury, Frequent kneeling and stooping
- clinical manifestations
Answer -joint pain most common
-Joint stiffness occurs after periods of rest or unchanged position -Early morning stiffness usually resolves within 30 minutes when moving around 1 / 2
-Overactivity -> mild joint effusion, temporarily increased stiffness -Crepitation -Asymmetrical -Systemic manifestations are not present in OA Distinction between OA and RA
- joint pain
Answer -Primary symptoms ranging from mild discomfort to significant disability
-Pain worsens with joint use
-Early stages
rest relieves pain
-Later stages
pain with rest and trouble sleeping due to increased joint pain
- deformities
Answer -Specific to involved joint
-Heberden's nodes (DIP joint) and Bouchard's nodes (PIP joint) -Red, swollen, and tender -No significant loss of function -Visible deformity (can lead to self esteem issues)
-Knee
bowleg, knock-kneed
-Hip
one leg shorter
- Herberden's nodes
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